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Pulmonary hypertension frequently exacerbates multiple myeloma through a variety of processes. These causes include concurrent cardiac amyloid-induced pulmonary hypertension, congestive heart failure, chronic thromboembolic pulmonary hypertension, high output heart failure caused by anemia or lytic bone lesions, and chronic thromboembolic pulmonary hypertension. This case study series demonstrates the numerous pathways via which people with multiple myeloma experience pulmonary hypertension. To determine the causes of pulmonary hypertension and how to treat it in patients with multiple myeloma receiving care at the University of California, San Diego. In order to identify individuals with multiple myeloma and pulmonary hypertension, a retrospective chart analysis was carried out. Patients also needed to undergo a right heart catheterization. Chart review provided information on pulmonary hypertension's demographics, comorbidities, clinical trajectory, and a etiology.
